Cognitive Behavioral Therapy

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Meichenbaum (1977) examined the increasingly important role assigned to cognitive factors not only challenges the traditional tenets of behavioral therapy but expands the highly specific procedures which have characterized in the field in recent years. The theoretical implications of increased interest in cognitive factors give direct attention to the nature of the client—therapist interaction, the content of inner speech and the client 's appraisal of outcome as active ingredients of the change process.

Ellis (1980) examined 32 important clinical and personality hypotheses of rational-emotive therapy (RET) and other modes of cognitive-behavior therapy and lists a large number of research studies that provide empirical confirmation of these
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Yet the overall strength of the association between cognitive distortions and externalizing behavior is unknown. In a subset of intervention studies that incorporated both cognitive distortions and externalizing behavior, however, neither cognitive distortions nor externalizing behavior were effectively reduced.

R. J., & Worrell, M. (2010) gave an research on need to challenge thoughts in cognitive behavioral therapy. It emphasizes the primacy of cognition in mediating psychological disorder. It aims to alleviate distress by modifying cognitive content and process, realigning thinking with reality.

Ilardi & Craighead (1994) gave this article about the role of nonspecific treatment factors in cognitive-behavior therapy (CBT) for depression. An analysis of relevant studies reveals that the majority of symptomatic improvement in CBT occurs prior to the formal introduction of cognitive restructuring techniques. Reviewed evidence supports a mediation role for the hopelessness construct in CBT. Two non specific factors, the treatment rationale and the assignment of homework, appear integral to early symptomatic improvement. The role of cognitive techniques is discussed in light of these
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A literature search was carried out for all brain imaging studies of patients with OCD and the basic science literature on the functional neuroanatomy of cortico-basal ganglia circuits. They concluded that OCD symptoms are mediated by hyperactivity in orbitofrontal-subcortical circuits, due to an imbalance of tone between direct and indirect striatopallidal pathways. The authors present a model which describes how frontal-subcortical brain circuitry may mediate OCD symptomatology, and suggest a hypothesis for how successful treatments may ameliorate symptoms, via their effects on circuit

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