Ophthalmology consultation was taken and it was cured in a week time by bandaging of eye. 3-4 mm septal perforation was seen in one patient. 6.66% patients showed synechiae formation which were later on excised under local anaesthesia. Some kind of residual deformity was seen in one-fourth patients, the most common of which was dorsum deviation in 20% patients, broad dorsum in 10% patients, residual hump and residual saddling in one patient each. Newer cosmetic complaints were recorded in 16.66% of patients.
Background Osler-Weber-Rendu syndrome, also known as hereditary hemorrhagic telangiectasia (HHT), is an autosomal dominant vascular dysplasia1, 2, 3. It is manifested by mucocutaneous telangiectases and arteriovenous malformations (AVM). Lesions can affect the nasopharynx, central nervous system, lungs, liver, gastrointestinal tract and conjunctiva. Recurrent epistaxis is the most common presentation. Diagnosis of HHT is made clinically on the basis of the Curaçao criteria: epistaxis, telangiectasia, visceral lesions and family history1.
The clinical examination revealed he had a bony hard, non- tender swelling, extending from the posterior border of the mandible upto tragus of the right ear, while the overlying skin was devoid of any secondary changes. (Figure 1 & Figure 2). The lateral oblique radiograph of the jaw revealed a unilocular radiolucent lesion extending from the distal surface of the first permanent mandibular molar, involving almost entire ramus encapsulating an unerupted tooth within the (Figure 3) ,with intact lower/inferior border of the mandible . As there was no cystic fluid available, provisional diagnosis of unilocular ameloblastoma was made. Routine blood investigation did not reveal any abnormal findings.
Stephen A. Rails, Gary R. Warnock in 1985 8 reported a 62-year-old male patient who had a varied medical history and oral lesions consistent with stomatitis areata migrans. The stomatitis areata migrans affected essentially all oral soft tissues, including the gingival tissues which were rarely involved. The gingival involvement was documented by clinical and histologic means. In our case the clinical appearance of the lesions remained consistent with SAM of the gingiva, with no concomitant dermatological problems or systemic diseases. Management Since the etiology was unknown the treatment is empirical.The patient was reassured as to the benign nature of the process.
Then the patient was subjected for Magnetic Resonance Imaging (MRI). On MRI scan, there was a large lesion composing vascular spaces which were seen extending into the floor of mouth and right parapharyngeal space. There was an exophytic component of the lesion causing partial obliteration of oral cavity. On Dynamic Contrast MRI there was gradual progressive contrast retention within the vascular spaces and no significant arterial feeders and early draining veins were noted (Fig 2 C-F). The imaging diagnosis of slow flow venous malformation was considered.
INTRODUCTION There are several types of congenital craniofacial anomalies, most frequent of which are orofacial clefts that encompass the cleft lip and palate (CLP), which occurs when embryonic facial processes fail to unite (1). The complications associated with CLP are maxillary growth aberrations and high occurrence of Class III malocclusions. In children with CLP, aberrations in number, size, shape, and period of tooth formation are more common than in the non-cleft population. Orthodontic abnormalities such as crowding, rotation, and malposition of teeth are also frequent in patients with CLP (2). ‘In the orthodontic context, an index is used to designate a rating or as a categorising system that assigns a numerical score or alphanumeric
7) SD-OCT showed diffuse deposition of hyperreflective material between the RPE and the Bruch’s membrane resulting in diffuse RPE elevation. Additionally, we observed a focal serous retinal detachment associated with hyperreflective dots in the interdigitation zone of the foveal region. The OCT-A choriocapillaris segmentation showed a high flow structure composed of filamentous linear vessels, forming anastomoses and loops, associated with a peripheral arcade surrounded by a dark halo. (Fig. 8) Case
Relapse in cavia cobaya models occurs rapidly. The left first insisivus compressed towards the distal side during 14 days orthodontic tooth movement and relapsed toward the mesial side. There was a rapid relapse initially following 2 days appliance removal Increasing osteoclast activity in tension side show that the number osteoclast situated along the alveolar bone mesial of insisivus at the end of active treatment. Decreasing osteoclast activity by applying Stichopus hermanii during relapse period means bone remodeling begins. Periodontal ligament plays a central role for remodeling periodontal ligament dan alveolar bone.
ABSTRACT Trisomy 18 is a common autosomal trisomy syndrome. It is due to either full or partial presence of an extra copy of chromosome 18. Its prevalence correlates positively with advanced maternal age. Affected infants usually exhibit a variable pattern of anomalies including growth restriction, marked psychomotor and cognitive disability and an array of physical findings including characteristic craniofacial features, clenched fists with overriding fingers, small fingernails, underdeveloped thumbs, short sternum and heart and kidney anomalies. The majority of these infants die within the first year of life; only 5% to 10% of them survive longer.