Rhabdomyolysis: an imbalance of toxins in blood due to the breakdown on skeletal muscle, could be just that. This disease can affect athletes of all ages and has devastating consequences. When, for instance a wrestler is hit hard on the mat and he injures a muscle, we normally wait for a bruise to arise and figure it will be gone quite soon. This impact on the muscle can actually lead to muscle damage or muscle death. The death of skeletal muscle tissue can leak poisonous intramuscular toxins such as myoglobin and creatine kinase (CK) into the blood stream and have you in the hospital within hours (Torres 2015). You at first, may think you are just tired after a hard hit , yet the swelling and weakness of limbs is much more dangerous than
Some of the databases that were searched for information about my PICOT questions were not directly related to patients in long-term care facilities. The Databases that were searched included CINAHL, National Guideline Clearing House, Ovid, and EBSCO, articles related to my PICOT was found on National Guideline Clearing House and Ovid. Some of the key words that were used during the search were Hendrich II fall risk tool, fall in Long-term care facilities, Geriatric falls, fall prevention and predicting fall. National Guideline Clearing (www.guideline.gov) produced about ten results directly related to the content in the PICOT question and CINAHL had thirty nine results.
Neurons transmit information to each other and to muscles, organs and glands. The nerve impulse is sent from the axon of one neuron to the dendrite of another neuron. The neuromuscular junction as labeled in Part A of this assignment, shows that there is a space between the axon of a neuron and the motor plate of the muscle cell. The two parts do not actually touch each other. When the football player’s brain sends a message to move during the game, the nerve impulse is sent from neuron to muscle cell. The space (synapse) between the axon of a motor neuron and a muscle cell is called a neuromuscular junction. The axon of the motor neuron contains synaptic vesicles which contains thousands of molecules of the neurotransmitter acetylcholine. This acetylcholine is released into the space between the axon and the muscle cell. The muscle cell membrane has receptors to accept or to bond with the acetylcholine. This region is called the motor end plate. When the acetylcholine bonds with the protein receptors in the motor end plate, sodium and potassium gates in the region open at the same time
In the autobiography, Laughing at my Nightmare, the author, Shane Burcaw, is diagnosed with spinal muscular atrophy, and the book deals with Burcaw’s life with spinal muscular atrophy. In order to understand spinal muscular atrophy, one must know the causes, the variations of spinal muscular atrophy, as well as the outlook of the disease.
The Edmonson Psychiatric Fall Risk Assessment Tool (EPFRAT) to have higher sensitivity in assessing fall risk in the geri-psychiatric population (Edmonson et. al, 2011). This project of EPFRAT will let the progress of a fall risk prevention protocol to provide the safest environment and best quality of care possible for the geriatric psychiatric inpatient.
Over the years, there has been various debates over how the elderly is being treated in long term care facilities. Each and every year the number of the elderly people living in the U.S continues to grow immensely and whether or not we have the right systems in place to deal with this overwhelming number of baby boomers remain questionable. This number is projected to continue to grow and it is important that the right care is provided and also that the elderly is properly taken care of as they make this transition. As the number of elderly people in long-term care facilities continue to increase, different and various issues arise. One of the many serious issues involved with the elderly in long-term care facilities is the use of chemical
There were several factors which may have contributed to this scenario. The patient’s comorbidities which include the ischaemic stroke which happened 2 years ago might have caused his fall. A Grade A recommendation and Level 1+ evidence were given by the National Stroke Foundation (2010) that patients are advised to undergo intensive rehabilitation for the first six months post-stroke. Given the fact that he had only received four months of inpatient rehabilitation, his functional status might not have been maximised. In addition, a Grade A recommendation and Level 1+ evidence were given for multi-disciplinary intervention in inpatient rehabilitation (MOH, 2008). However, the inpatient rehabilitation approach in the patient’s holistic management is
The elderly in nursing home are at an increased risk for falls, more than any other area. An average of 5% of all persons over the age of 65 reside in nursing homes. On average, over 1,800 people over the age 65 fall while in nursing homes every year and 20% of all fall related deaths occur from falls while living in nursing homes (Centers of Disease Control & Prevention, 2012).
This injury is the stretching of ulnar nerve that runs through cubital tunnel, which is is the largest unprotected nerve on the body. The nerve can tear when the ulnar nerve is compressed. It is between the medial epicondyle and olecranon, and runs along the ulnar bone. For the muscles, the cubital tunnel is adjacent to to triceps and continues down the forearm between the flexor carpi ulnaris muscle.It can be either chronic or acute, but it typically acute. It is near the medial head of the tricep and the arcuate ligament. Also, the arcade of struthers, medial intermuscular septum, and the deep flexor aponeurosis are affected.
Amyotrophic lateral sclerosis is a degenerative neuromuscular disorder that affects the motor neurons of the spinal cord and brain. Due to degeneration or destruction of the motor neurons, muscles throughout the body begin to become weak and waste away to the point that an individual has no muscle movement. In most cases, due to having muscle strength, a patient with ALS will succumb to their disease because of respiratory failure or dehydration and malnutrition. It is a progressive disease with a prognosis of 3 to 5 years after initial diagnosis. There is no cure for the disease as of now. This paper will discuss a more in depth look into the symptoms, diagnosis process, treatment options, medications, prognosis, and ongoing research.
Getting out of bed is one of the dangerous things that the elderly patients do when they are admitted in the hospital. Study conducted by Ambrose, Paul & Hausdorff, (2013) on patient falls reveals that a majority of falls in the elderly patients occur between 0700 and 1900, especially when they are getting out of bed to use the rest room. The cause of their falls is mainly due to unsteady gait, memory loss, confusion that comes with age.
Dr. Marshall Westwood had a meal of puffer fish and rice for dinner in Indonesia on his recent trip. Within an hour, the numbness starting from his lips and tongues quickly spread out through his face and neck, and he had pains in stomach and throat with symptoms of severe nausea and vomiting.
Living with chronic illness and pain is something that isn’t easily understood by people who are not going through the same thing. Moreover, the effects of illness are an emotional and personal daily battle. All of us with Fibromyalgia are not the same!! Not one of us has the exact same symptoms. We have a lot of the same, some are worse, some are less, but no two are exactly alike!
Myasthenia Gravis as well as known by the name (MG) is autoimmune neuromuscular disease that caused weakness in the skeletal muscles, the muscle that are used for movement to do activity of daily living. Myasthenia Gravis are caused by the neuromuscular disorder. The neuromuscular disorder for Myasthenia Gravis occurs when communication between nerve cells and muscles becomes impaired. This neuromuscular disorder caused the important muscle to make the contraction movement from occurring, that caused muscle weakness, resulting rapid fatigue of any of the muscles under voluntary control. Symptoms include drooping of one or both eyelids, diplopia and difficulty swallowing, chewing and speaking. There were no cure for the disease
There are four steps in the muscle contraction cycle. At the beginning of contraction, the sarcoplasmic reticulum releases ca2+ into the sarcoplasm where they will bind to troponin.Troponin then moves tropomyosin away from the myosin binding sites on actin. Once binding sites are free the contraction cycle will begin. Step one of the contraction cycle is ATP Hydrolysis. During ATP hydrolysis the enzyme ATPase uses water to cleave a phosphate from ATP producing ADP and a free phosphate which remains attached to the myosin head. The energy that was released from breaking the chemical bond is used to move the myosin head into position for attachment to the actin molecule. Step two of the contraction cycle is Cross-Bridge formation. During cross bridge formation the myosin head attaches to the revealed myosin-binding site on actin forming a cross bridge between the two protein molecules. Step three of the contraction cycle is the power stroke. During stage three the release of ADP and P from the myosin head triggers the power stroke, the rocking of the myosin head pulls actin towards the M-line. The fourth stage of the contraction cycle is the detachment of myosin from actin.The end result is a shortening of the sarcomere,the distance between the Z discs shortens, the H zone disappears,the dark A band increases because the actin and the myosin overlap more, and the light I band