Diabetic eye disease includes diabetic retinopathy, cataract and glaucoma. What is Diabetic Retinopathy? Diabetic retinopathy is one of the complications of diabetes out of tripathy. Diabetic retinopathy occurs when hyperglycaemia (high blood sugar levels) causes damage to the cells at the back of the eye, known anatomically as, the retina. Diabetic retinopathy is a leading cause of loss of vision.
Symptomatic patients presenting with a palpable volar swelling should be examined for variations in the palmaris longus. In an asymptomatic subject a hypertrophied muscle will obstruct and result in hazy interpretation of radiological assessment of this region. Difficulties in endoscopic procedure and electro myographic studies may also result from such a reversal
Patients often experience headaches that are bilateral and located in the frontal temporal region. It is associated with pulsating pain, nausea, photophobia, phonophobia and aggravated with activity. (Pathostatement) The pathophysiology is often elusive. The cause is often neurovascular and take place in the brain. A type of neuron event can cause an imbalance in the trigeminal system causing inflammation.
Polyclonal B-cell hyperreactivity with abnormalities of autoregulation have been reported, the histopathologic substrate of Sjogren’s syndrome is characterized by presence of lymphocytic infiltrates in glandular and extraglandular sites. Microscopic examinations of enlarged parotid or submandibular glands reveal benign lymphoepithelial lesion, characterized by epimyoepithelial islands in a lymphocytic infiltrate with replacement of acini. In the minor salivary glands the characteristic feature is focal lymphocytic
In addition, diabetic neuropathy is known to have a profound impact on patients sleep, mood, daily activities and quality of life. Therefore, it is very important to establish a proper therapeutic option to prevent or decrease the incidence of diabetic neuropathy. At present there are many drugs used to treat Peripheral Diabetic Neuropathy, but many of these drugs has some side effects such as, dizziness, drowsiness, nausea and
In adults, this primary infection is more severe and in immunocompromised patients, it can be followed by complications such as, high fever, pneumonia, encephalitis and hepatitis (Gershon et al., 2013). During this primary infection, the virus can be
But none of the asymmetric circuity has been found nor have their roles in learning and memory been studied. With the single neuron connection prediction and validation, the function of the first, comprehensive asymmetric neuron network can be further tested with different manipulations such as short-term, long-term memory training, or down/over expression of memory proteins in specific neurons. 2.3 Timeline (Yearly goal) Year 1: Use Flycircuit database to identify asymmetric circuit candidate in the central brain and their neuronal polarity, search for specific driver for labelling asymmetric neurons establish their neuron types (neurotransmitter). Furthermore, record functional responses from asymmetric neurons upon simple stimuli such as odor or light, with GCaMP. Year 2: Record functional responses with training process in Drosophila such as odor-shock pairing and odor-reward pairing.
INTRODUCTION Research Question To what extent is genetics responsible for causing Systemic Lupus Erythematosus? Foreword Systemic Lupus Erythematosus is a chronic autoimmune disease wherein the immune system mistakes healthy cells as intruders and attacks them. These attacks could happen in various parts of the body. It is most commonly known as the ‘great imitator ‘as its signs and symptoms imitate that of other diseases. Lupus differs from each patient and symptoms oscillate from time to time.
Botulinum Toxin for Hyperhidrosis Treatment Abstract Primary focal hyperhidrosis is a disorder of excessive sweating which is typically of unknown etiology. Axillae, palms, soles and face are typical area for primary focal hyperhidrosis. The disorder is often overlooked, but its effects on social and occupational lives can become a big hassle for many people in everyday life, particularly in severely affected people. This also leads to emotional and psychosocial distress. Certain treatment strategies, such as topical formaldehyde, iontophoresis and oral anticholinergics have been used in clinical practice for quite a while, yet mostly come up with adverse effects and complications that eventually unable to meet patient’s satisfaction.
NAME: Cassianna Sookram ID#: 812003173 DATE: 20.04.2016 CASE REPORT: Retinitis Pigmentosa (Pathology 2) ABSTRACT This case looks at the clinical findings, causes, signs, symptoms, diagnostic tests and management options of a patient with retinitis pigmentosa. Keywords: Retinitis Pigmentosa INTRODUCTION Retinitis pigmentosa (RP) is an inherited, degenerative eye disease that causes severe vision impairment due to the progressive degeneration of the rod photoreceptor cells in the retina. It may be inherited as autosomal dominant (seen often), recessive or X-linked. It must be noted that RP can also occur as an isolated sporadic disorder and it is associated with myopia. This disease has no preference for age, it can occur to anyone from early
Temporal lobe epilepsy (TLE) is a type of epilepsy characterized by recurrent, unprovoked seizures that originate from the temporal lobe structures (Ko & Benbadis, 2014). The temporal lobes are important for memory, hearing, and language (Kuzniecky & Jackson, 2005). TLE is the most common form of focal epilepsy (Jutila et al., 2002). TLE is also the most common type of epilepsy requiring surgical treatment (Janszky et al., 2005). The seizures associated with TLE consist of simple partial seizures and complex partial seizures.