Patient's Case Study: Syncope

This can lead to heart and damage the heart muscle permanently crisis; • high blood pressure, which is also called hypertension; • disease of the heart valves - there may be a leak in the valves or those may stiffen, especially the sigmoid valves and mitral; • a disorder of cardiac muscle, which is also called cardiomyopathy. That may be caused by a virus, bacterium or other infections; • damage to the heart caused by the abuse of alcohol or drugs; • congenital heart disease and heart defects that exist at birth; • an irregular heartbeat, especially if there is rapid or chaotic. The heart does not pump enough blood when his rhythm is abnormal; • certain lung disorders, such as pulmonary hypertension, which occurs when the right ventricle dilates, causing heart failure on the right. Normal Heart congestive heart é START T How is the diagnosis?

Monitor the heart rate and pattern Mr. Roberts has already developed sinus tachycardia with short runs of ventricular tachycardia, ST-segment elevation, T-wave inversion, and the development of Q waves over most of the anterior V leads on his electrocardiogram. The ST-segment elevation and the T-wave inversion indicate a possible Myocardial Infarction The low serum levels of potassium due to fluid shifting back to the intracellular compartments, the myocardium excitability increases resulting in tachycardia and abnormal EKG patterns Monitor fluid status Weight the patient daily

This is a 25 year old African American male who is here because he is experiencing burning secsation with urination, and irritation at the penus. Patient is also requesting stuture removed form his right hand. difficulty with Patient denies chest pain, SOB, N/V/D, or fever. Patient denies depressive moods, thoughts of suicide or homicide. current pain

Later during discussion with the team to rule out the reason of Brady arrest, patient history was reviewed. Through that I came to know that patient came with the complain of chest pain and SOB in ER .Initially angina protocol was conducted and patient was diagnosed with NSTEMI according to the 2010 AHA guide line .After initial diagnostic procedure and after initial management, patient was shifted to CCU for further management .while reviewing patient 24 hours condition .I was identified that in routine ECG .There was ST elevations in inferior leads that is T wave inversion in leads 2 ,3 and AVF .This

If a patient cannot tolerate ACE inhibitors medication, vasodilators are also an option (Macon B.). Beta-Blockers can help reduce the blood pressure and slow down the rhythm of the heart (Macon, B.). Since a heart failure may cause the body to have more fluid than it should, diuretics may be used to reduce the fluid content in the

Many problems can lead to lack of blood some include: problems with the lungs, airways leading to the lungs, heart problems, drug overdose, and exposure to cold water or air. One is “Tetralogy of Fallot, Which is the most common cyanotic heart defect seen in children beyond infancy. Tetralogy of Fallot is also the most common cyanotic congenital lesion that is likely to result in survival to adulthood and thus is the most common complex lesion to be encountered in the adult population after repair. The original anatomic description of tetralogy of Fallot included a tetrad of malformations: ventricular septal defect, right ventricular outflow tract obstruction, aorta overriding the ventricular septum, and right ventricular hypertrophy.” (Jacobs,

This type of shock occurs when the heart is unable to pump blood effectively. This is evident to patients who have had myocardial infarction, such as John’s case. In this illness, the heart has decreased contractility resulting to decreased cardiac output. Such decrease will stimulate the sympathetic nervous system to activate the compensatory mechanism by increasing the heart rate as evident in John’s vital sign to increase the peripheral pressure and ventricular

Assessment 2 Short Essay Question -01 Discuss Mr. Ronald bates systemic assessment and priorities of management Mr. Ronald bates presented to the emergency department with shortness of breath (Respiratory rate- 24 breaths/min) and general discomfort (pain score- 4/10) and it was started in the morning and worsens when doing activities. The above presenting complaints lead to a possible cardiac event, so that this presentation would be triaged as category 2. Therefore, medical officer would be notified regrading patient presentation and put Mr. bates to semi fowler’s position in the Emergency bed if this position is comfortable for him. Further primary systemic assessment of the patient starts with an order with an assessment of

But the staff understood and Dr. Westwood got an ambulance and reached to ED. He presented with diaphoresis, motor dysfunction, paresthesia, nausea, and ascending paralysis from his leg to the upper body, arms, face and head. He became cyanotic and hyperventilating and it turned to be bradycardiac with a BP 90/50mmHg. After five hour long clinical treatment procedures were followed for tetrodotoxin poisoning, his vital signs were

The patient appears to be alert, engaging, and oriented. There was no evidence of

An ECG reading showed that Mr Collins had developed sinus tachycardia. He was anxious, and tachypnoea. Peripheral pulses (except the brachial pulse) were absent. Capillary refill time was 7-8 seconds. Mr Collins’ skin was cool and clammy.

Pupil equal and react bilaterally to light. Hair dry and clean without scalp. Ears clear without redness and swelling, no hearing aids, medium amount of cerumen in ear canal. Nares redness with small drainage. Report the finding to charge nurse Dentition

The patient for the purpose of this essay will be referred to as

Pulmonary: Chest wall symmetric, respirations even and unlabored. Lungs CTA. Cardiac: Sinus tachycardia, NL S1, S2. No murmurs or rubs.

The patient is a 63-year-old woman who presents for follow up after hospitalization at [Place], because of GI bleeding. The patient had presented here after years of living away, complaining of a history of progressive bloody diarrhea. Lab work was markably abnormal with significant anemia, with hypoalbuminemia, hypokalemia. The patient underwent a full evaluation, and was found to have colitis. She was treated with IV steroids, and has noted marked improvement in her condition. At the time she became ill with her diarrhea her blood pressure went down, and she discontinued antihypertensive. Now that she has improved, she has noted that her blood pressure has been going up, and she did resume her medications. It is unclear that she needs to resume them both, at the time of her discharge from the hospital on prednisone.