ARTICLE REVIEWED: -by vignesh balasundaram
Is a Cancer Diagnosis Associated with Subsequent Risk of Transient Global Amnesia?
JianweiZhu1, 6*, DonghaoLu1,Olafur Sveinsson2, KarinWirdefeldt1,3 KatjaFall4, FredrikPiehl2, UnnurValdimarsdóttir5, FangFang1
The whole article describes the situation in which the cancer diagnosis process may lead to transient global amnesia {TGA}. Transient global amnesia is a neurological defect which is temporary but may involve total disruption of short-term memory which could be either retrograde or anterograde which is either past memory or the newer one. It is common in age group of 50-80 years and an incidence of 5-11 per 100,000 individuals. The condition
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The research yet seems to become a failure that cancer diagnosis most probably not associated with transient global amnesia. It failed to provide sufficient results to prove the above mentioned concern. Also the article failed to provide the reasonable cause for transient global amnesia. Still the etiology of the defect remains unknown. Though it is a failure, the article correctly describes the epidemiology behind the survey because, that was the first research ever to relate cancer diagnosis and transient global amnesia. The article describes cancer diagnosis may lead to severe stress which may give rise to transient global amnesia, but the reason for connecting the TGA with stress is not properly mentioned in the article, starting the survey with a hypothesis. The article correctly describes the hospital-based evidences which are required for the evidence-based study and those studies showed some sort of prevalence. The aim of the project and the survey ended up in a different discussion that transient global amnesia is commonly …show more content…
The ascertainment both cancer and transient global amnesia are clearly given in the article. The hospital-based evidences are not too wider. Though the data were lot, the result of the article didn’t relate the aim of the article. Above all the article has a wide data which may be useful for any other studies. The discussion of the article defines that both physical and mental stresses may lead to transient global amnesia but only based on evidence. The survey was also only under a small range of population. But when went for five million Swedish population, but gave negative results. The identification of TGA in the study relied on hospital administrative records (i.e., inpatient and outpatient hospital visit), and as a result they were only able to identify TGA cases admitted over-night in a hospital or treated by a hospital-based specialist. The strength of this study is a large scale cohort study. The validation of hospital-based study was taken only from two hospitals which is not sufficient enough. The statistical analysis also included incidences among various types of cancer such as prostate cancer, breast cancer, etc. After excluding certain factors such as economic
With this loss, the brain becomes a station of short facts that are not remembered or greatly learned. Noteworthy changes in the way we take in knowledge like this have been happening throughout all
At the time the physician removed parts of his hippocampus as a way to stop his seizures. HM seizures went away, but he could not form any new memories. However, one form of memory left intact in both HM and Leonard is the ability to learn skills. This ability is known as procedural memory, a memory that is shown by performance rather than by conscious recollection (Kalat, 1998), it allows us to learn how to do things such as ride a bike or play an instrument. The areas of the brain outside of the hippocampus are involved in procedural memory, which is why an injury that results in anterograde amnesia doesn't affect procedural memory.
Anterograde amnesia is when patients can remember new non-declarative (implicit) memories, but have a shortfall when forming new declarative (explicit) memories. Further,
(2007) all underwent neuroradiological and neurophysiological exams to determine the presence of brain damage and confirm the form of amnesia they had. The results were consistent in that the case study with PA showed no brain damage while the OA case studies did show brain damage in the frontal lobes and hippocampus. Yet, only one PA case study is looked at while there are two OA cases in the Serra et al. (2007) paper, this isn’t a large sample. A problem encountered with finding case studies to compare is that individuals suffering from the amnesia being investigated can be rare and so finding larger samples is very difficult.
The thalamus, hypothalamus, amygdala, and the hippocampus are structures in the brain that enables us to remember, recall and creates new memories. Wearing showed specific symptoms of his memory loss. He would repeat himself often and exhibit moment to moment consciousness. He often showed extremely emotional behavior like fits of rage. After a period of about two minutes he felt that he was awake for the first time,
Anterograde amnesia is a brain injury that can effect one 's ability to recall events or memories of what happened. When a person is having trouble remembering things they will often say that I can 't recall what I saw and I know it but it is not there. When dealing with an Injury or illness this Anterograde amnesia can take place after a tragic event and leave the person feeling like everything is feeling slower. There are symptoms that came with anterograde amnesia like learning to cope with new information they received.
The biological approach to the basis of memory is explained in terms of underlying biological factors such as the activity of the nervous system, genetic factors, biochemical and neurochemicals. In general terms memory is our ability to encode, store, retain and recall information and past experiences afterwards in the human brain. In biological terms, memory is the recreation of past experiences by simultaneous activation or firing of neurons. Some of the major biopsychological research questions on memory are what are the biological substrates of memory, where are memories stored in the brain, how are memories assessed during recall and what is the mechanism of forgetting. The two main reasons that gave rise to the interest in biological basis of memory are that researchers became aware of the fact that many memory deficits arise from injuries to the brain.
In the article “Memory Distortion in People Reporting Abduction by Aliens” the Researchers are looking at how memory distortion effects people who report recovered memories of traumatic events that seem unlikely to occur. The researchers first examined recall and recognition in more likely events such as childhood sexual abuse and from the findings created four hypotheses to test on their experiment about memories of alien abductions. In the experiment they created three groups of individuals in which to test their hypotheses. Once the groups were selected the researchers used the Deese/Roedier-McDermott paradigm to test the groups false recall and false recognition. After this the researchers concluded that two out of their four hypotheses
False memory syndrome (FMS) is hypothesized to be correlated with poor experiences regarding mental therapy or treatment and social pressures which can contaminate memories while in REM-sleep. Common social pressures include: body image, tradition, religion, and marriage; the Influences listed may be capable of distorting memories into any direction whether it be a memory of abuse or non-abuse. Knowing the malleability of memory is key to understanding how to shape FMS memories. The alteration of FMS memories can be a vital asset when aiding psychotherapy patients in recovery from traumatic events. Sleep deprivation has a correlation to weakened memory consolidation.
They are often seemed to be feeling confused or disoriented. This disease unlike Alzheimer can occur at any age. And thus can lead to a partial or full Amnesia disorder. In few cases patients are never left unsupervised too as they may harm themselves.
The patient had a full recovery after 11 ECTs. It was found that ECT became a very popular treatment because it was cheaper and could be very effective with 10-20 treatments. A side effect of ECT was retrograde amnesia, so the patients
Damage to the hippocampus area of the brain can lead to anterograde amnesia. Anterograde amnesia is the inability to form new long-term memories. Declarative (explicit) memory is a type of long-term memory that contains information that is conscious and known. This information is things that people can know. Semantic memory is a type of declarative memory containing general knowledge, such as language.
It is that they have forgotten and needs something to bring forth these memories as the brain can only store a certain amount of information at a time and to
The Matrix in Your Head Extensive research has shown the important role of the hippocampus in our ability to store memories in a spatial context. This spatial sense is not only related to time, but also to the correspondent physical place where memories were created. The results of the study performed by John O'Keefe and Jonathan Dostrovski in 1971 have further proved this concept of a “cognitive map.” One of the first evidences of the role the hippocampus plays in storing our memories was established in the 1950s, when surgeon William Scoville removed most of the hippocampus of one of his patients in an effort to save the patient's life.
There has been considerable recent interest in the phenomenon of false memory and its implications in clinical, legal and laboratory settings. This interest has prompted several researchers to investigate the neural mechanisms underlying illusory