The most common treatment for these infections, caused by Staphylococcus aureus is the antibiotics. There are many kinds of antibiotics using in the modern days, but the first kind of antibiotic being introduced for treating Staphylococcus aureus was Penicillin in 1943. This kind of antibiotic stops the formation of peptidoglycan cross-linkages that makes the bacterial cell well stronger. This eventually makes the cell wall formation and degradation become imbalanced, consequently lead to the cell to die. Other kinds of antibiotic were quick introduced for treating Staphylococcus as well.
But what do the antibiotics exactly do? And how they can cure diseases? As a matter of fact, Antibiotics are powerful medicines that can cure bacterial infections if they are used properly. They fight against bacteria by destroying or inhibiting bacteria growth. Soil bacteria and fungi are the natural components of Antibiotics ((n.d.).
Antibiotic medications have saved many people’s lives. Unfortunately antibiotics are no longer as effective in stopping pathogenic bacteria infection. Currently there is an antibiotic resistance crisis since may bacteria have become or are becoming resistant to all of the antibiotics developed. Instead of researching new antibiotics, which will continue to promote antibiotic resistance, antibiotic stewardship should be promoted specifically dealing with preventing infections, monitoring antibiotic prescription, and developing laboratory tests that provide accurate results faster. Hospitals need to reduce the infections contracted during patients stays in their facilities.
First of all, the defected bone marrow must be destroyed which arises a huge risk to new diseases. Healthy stem cells are then introduced and the patient is given time to recover where blood counts are monitored
Drugs produced in labs can have dosage forms changed quickly i.e. how the drug is delivered. For example, tablet or capsule form. 6.2.1 and 6.2.2 Penicillin An antibiotic in the penicillin group of drugs helps fight bacteria in the body.
meningitidis has extensive mechanisms to elude the host immune system, of which tactics to evade complement deposition are of major importance. The main mechanism is the recruitment of a human negative regulator of the complement system, fH, by the membrane protein fHbp. This binding, by preventing the assembly of C3 convertase, inhibits the deposition of complement on the bacterial surface and increases its chances of survival. In addition, N. meningitidis can exploit host weakness by upregulating the expression of proteins involved in immune evasion in response to elevated temperature, as is the case in fever. Future research should seek to target specific antigens on the surface and inhibitors of the fHbp-fH interaction to be able to treat or prevent this disease that can have severe
Tetracycline which is used to treat acne, respiratory tract infections and other conditions kill bacteria by inhibiting protein synthesis. This is done by stopping molecules to bind on a certain area of the cell called ribosome. Since key molecules can bind to ribosomes which is the site where protein synthesis happens the bacteria will die because it can’t carry out vital functions including asexual reproduction. Some antibiotics kill bacteria by stopping the production of folic acid which is an essential vitamin for proper functioning of the cell’s
The treatment of oral Chlamydia is very quick once it has been diagnosed. The usual prescribed treatment for oral Chlamydia is a single dose of antibiotics or a week-long course of antibiotics. The two most common antibiotics prescribed for oral Chlamydia are: • Azithromycin. This is a prescription drug used to treat infection caused by bacteria. Azithromycin works by killing bacteria such as Chlamydia trachomatis and as well treat oral Chlamydia and other sexually transmitted infections caused by bacteria.
In addition to plasma, red blood cells, and white blood cells, there are platelets. Platelets are disc-shaped cell fragments that prevent a great amount of blood loss from cuts by developing platelet "plugs," as their granules contain chemicals that make blood clot (Ballard, 33). What is blood pressure? " When the ventricle of the heart contracts, blood is pumped into the
aeruginosa’s ability to mutate genetically and transform into different antibiotic-resistant strains, make it the most important bacteria species studied among patients suffering from CF. It’s ability to form a glycocalyx and prevent the host’s white blood cells to attach and phagocytize the pathogen also contributes to its importance of a pathogen. The formation of a biofilm communicates to the bacterial colony that it is being attacked by antibiotics and to mutate to adapt and survive. The mutation of the bacteria to form an alginate to protect it from environmental stresses as well as the ability to release powerful exotoxins to inhibit protein synthesis of the host’s cells add to P. aeruginosa virulence and ability to develop chronic lung infections in CF
Lockjaw, also more commonly know as tetanus or Clostridium tetani, is a desease caused by bacterial infection. the bacterium Clostridium tetani, is commonly found in soil, saliva, dust, and manure and can be introduced to the blood stream via a skin puncture, the bacterium produces a toxin called Tetanospasmin which interferes with muscle contractions, resulting in muscle spasms or spasmodic muscle contractions. (https://en.wikipedia.org/wiki/Tetanus). Tetanospasmin permanently binds receptors, so in that sense it can 't be cured. In the case of advanced tetanus treatment involves antibiotics and paralytics while being put on a respirator, which is needed until your receptors regenerate.(Andrew Lehman, Immunologist and Virologist, https://www.quora.com/How-can-tetanus-be-cured).
According to the AAP, a shiny nail is just as likely to give you tetanus as a rusty one. Clostridium tetani are the bacteria to blame for the Tetanus disease. The Encyclopedia of Human Diseases and Conditions states that the infection begins when the bacteria spores become active after moving deep within the body. Once the tetanus spores are active, they begin producing a toxin that attaches to the nerves around the area of the wound. The tetanus toxin spreads to the ends of the nerves of the spinal cord where they meet the muscles, resulting in severe muscle spasms powerful enough to tear muscles apart.
Major Quest Outline Name: Madison Underwood Thesis: Bacteria is becoming resistant to antibiotics and will eventually cause an apocalypse. Body: I. Understanding antibiotic resistance is vital in understanding the argument. A.
When affected, the cell can 't perform its basic functions correctly and harms itself. It primarily targets liver, lymphoid organs, which protect the body from infection, and kidneys. If one does