Gilbert Syndrome
Gilbert syndrome (also called Gilbert's syndrome) is a common condition where the liver does not process bilirubin, a substance that is produced from the breakdown of your red blood cells. Gilbert syndrome is typically harmless and requires no treatment.
Part 1: What Is Gilbert Syndrome?
Gilbert syndrome is characterized by elevations of unconjugated bilirubin in the blood, which is usually unrecognized until it is seen in a blood test that may be done for other reasons. Fluctuations in bilirubin levels in the blood (or hyperbilirubinemia) that are sometimes associated with yellowing of the skin and eyes occur in people who are diagnosed with Gilbert syndrome. This mild condition, which is otherwise harmless, is often recognized
…show more content…
These include:
• dehydration
• fasting state
• an infection
• stress
• physical exertion
• lack of sleep
• after surgery
• menstrual periods
Avoiding some known triggers can help reduce having episodes of jaundice.
Part 4: How to Live with Gilbert Syndrome
Gilbert syndrome does not require treatment. Although your bilirubin levels may fluctuate and you may experience jaundice occasionally, this usually disappears on its own, and does not cause any problems.
Gilbert syndrome is a long-term condition that is not a threat to your health. It is not associated with any complications and it does not increase your risk of liver disease. Symptoms are usually short-lived and self-limiting.
There is no need to modify your diet or your daily activities, except to avoid those that trigger your symptoms.
People with Gilbert's syndrome, however, may be at risk of getting jaundice and other side effects when taking certain medications such as:
• Indinavir and Atazanavir (used for treating HIV infection)
• Statins (cholesterol-lowering drugs) plus gemfibrozil
• Gemfibrozil (another cholesterol-reducing drug)
• Irinotecan (used for treating bowel
Hypoprothrombinemia caused by vit K malabsorption Adult: PO/IM 2.5-25 mg, may repeat or increased to 50 mg, Child: PO/IM 5-10 mg, Infant: PO/IM 2mg. Hypoprothrombinemia caused by oral anticoagulants Adult and child: PO/SC/IM
Clinical manifestations that indicate the presence of cirrhosis include the following: fatigue, ascites (fluid accumulation in abdomen), drowsiness and hepatic encephalopathy (Cleveland Clinic Staff, 2014). Room 362’s chief compliant of weakness and drowsiness was displayed upon admission and provided
What is the normal fate of bilirubin, and what role does the liver play? Explain how Mrs. Fender’s cirrhosis is related to her jaundice. • Bilirubin is a product of the heme of hemoglobin formed during the breakdown of erythrocytes. The liver removes the bilirubin from the blood and excretes it into the intestines as bile. When the liver is damaged, bilirubin, which is a yellow pigment, spills over into tissues and the blood, thus giving the skin a yellowish coloring.
He does have a slight elevation in his total bilirubin is 0.3. Other liver function testing is normal. He has previously had a liver ultrasound back in October of 2014, showing hepatic steatosis, otherwise negative abdominal ultrasound. He has previously has declined evaluation by gastroenterology or hematology and he is still not sure that he wants to do that. He is not having any belly pain.
Christina George #109193966 HAN 312 FALL 2015 CASE STUDY PROJECT PART 1 This assignment is due Friday 10/23/2015 by 9 am 1. 5 POINTS: Briefly explain the disease or disorder you picked in no less than five but no more than 10 sentences • Systemic lupus erythematosus (SLE) is a chronic autoimmune disorder that can affect any organ in the body. Symptoms range from mild to severe.
He is also a contributing author to a number of textbooks in the area of skin disorders and burns. When asked about an example of a common healthcare problem that he sees, Dr. Fagan said “Stevens-Johnson Syndrome (SJS) and toxic epidermal necrolysis syndrome (TENS) are two of the most common types
Amber lives in her smoke free home with her boyfriend and two dogs. She has been diagnosed with Vigitlio and is being treated for this condition by Dr. Harris at the University Medical Center in Worcester. Amber was told due to this condition is the reason why she suffers with neuropathy, arthritis, bursitis and fibraliga. This disease is progressive and her condition will worsen over time, which brings on muscle weakness and difficulty with coordination.
But, if you leave your Addison’s disease untreated, you may have an addisonian crisis which is caused by stress, infection, and injury/illnesses. This crisis results in low blood pressure and sugar, and high potassium levels. But, this can
No known food, drug, or activity can cause Hemangioma. Hemangioma is a collection of extra blood vessels in the skin. The symptoms for this oversized birthmark symptoms are: nausea, vomiting, abdominal discomfort, loss of appetite, unexplained weight loss, and a feeling of fullness in the abdomen. Treatments are a gel with the drug timolol applied to the affected skin, Corticosteroid medication, and Laser
What are the primary pieces of evidence from the CBC that point to this diagnosis? Anemia is a condition that develops when a person’s blood lacks enough healthy RBC or hemoglobin. The effect is an insufficient amount of oxygen to the tissues. In Harold’s case, the main parts of evidence from the CBC include the low RBC, hemoglobin, and hematocrit.
Also, blood disorders, diabetes, pale skin, sun damage, and even certain medications can cause the blood vessels to weaken. A way to prevent bruising on the skin is to see what medications that weaken blood vessels and eat vitamin K rich foods like kale and spinach. The disorder effects the integumentary system as it is a weakened spot on the skin. The skin is the first line of defense
The copper is normally absorbed at first, but it is not incorporated into caeruloplasmin and the excretion of copper within bile is reduced. An increase in the concentration of copper found within the cells of the liver causes excess copper to flow into the bloodstream; this increases the concentration of copper found within the plasma. Having the increased concentration of copper leads to a decrease of copper excreted through feces, and an increase in the concentration of urine. Wilson’s disease often proves difficult to diagnosis at first because its symptoms mimic those of other hepatic disorders, and don’t present themselves until adolescence (around 12-23 years of age). For instance, the accumulation of copper in the brain may result in neurological/psychiatric symptoms.
Klinefelter syndrome, also known as ‘47,XXY’ and ‘XXY’ is found in males, this is due to the fact that the host male gets another X chromosome. The image on the right you can see the extra chromosome with the pair of sex chromosomes. Usually there are only two chromosomes that determine the sex, one from opposite sexes but when it comes to Klinefelters Syndrome there is an extra X chromosome. Because this due to the additional chromosome it can described as a chromosome disorder.
Excessive bruising This symptom may be an indication of some abnormal activity of red blood cells, which in turn often leads to leukemia. Bruising in strange places, like fingers and palms and red spots on the face, neck, chest, symptoms that you must not ignore them. 7. Pain in the abdomen or pelvis
It provides a clear description of what CHS is and how it affects someone with the syndrome. Brewerton and Anderson take the reader through the full process of someone with CHS. They start by presenting the reported symptoms, the diagnosis, and the treatments for the syndrome. The authors also discuss the pathophysiology and the different theories of CHS. The purpose of this case report is to provide the facts on CHS and what causes the syndrome.