Variability of Digital Flexion Creases in a Population group of West Bengal Sangeeta Dey*1 and A.K. Kapoor2 1UGC-JRF, Department of Anthropology, University of Delhi, Delhi – 110007, India. 2Professor, Department of Anthropology, University of Delhi, Delhi – 110007, India. *Corresponding author: Sangeeta Dey, UGC-JRF Research Scholar, Department of Anthropology, University of Delhi, Delhi – 110007, INDIA. Email: 1sangitadey29@gmail.com 2anupkapoor46@rediffmail.com Abstract: Digital flexion creases occurs on the volar surface of each finger at the level of interphalangeal joints. Three digital flexion creases are normally observed to exist on each finger of the hand except for the thumb because of its different anatomy; distal inter-phalangeal …show more content…
They are also regarded as one of the external anatomical landmarks of the hand (Singh et al., 2002) in addition to palmar, thenar and wrist creases. According to Wurth (1937) and Aue-Hauser (1979), these creases are not formed primarily by embryonic flexion movements. They arise independent of fetal palm movement and are genetically determined. The presence of extra, displaced and missing digital flexion creases in individuals with normal joint anatomy and unusual digital creases variants in a number of syndromes indicates that genetic factors do probably play a role in digital creases formation (Stevens et al.,1988; Kamath et al., 2002). There are numerous studies focusing on different variants of flexion creases which are featured in a number of syndromes such as in Allagile syndrome (Kamath et al., 2002); partial deletions of chromosome 1q (Watson et al., 1986); Sickle cell anemia (Zizmor, 1973); Down’s syndrome (Penrose, 1931; Plato et al., 1973). However, few systematic studies are available on incidence patterns of digital flexion creases. This paucity of literature has prompted the author to conduct this preliminary work which aims at documenting the prevalence pattern of all the flexion creases …show more content…
The occurrence of triple creases is minimal in the distal and proximal phalanx. Digit III is characterized by the highest number of extra creases at the proximal and metacarpo-phalangeal areas followed by digit IV then digit II and lastly digit V. Similar results were encountered among Japanese by Aue-Hauser et al (1980). A comparison of the creases patterns between right and left hand (Tables 1 and 2) reveals that there is no significant difference with respect to handedness. However mono and double creases tend to be more frequent in right than left hand while triple creases are encountered more in left as compare to right hand. The difference observed between the sexes was also statistically non-significant. Although, there is a uniform tendency that males have more low numbered creases than females however there is a predominance of high numbered and extra creases among females. Extra+ creases shows higher incidence among males. The typical patterns of flexion creases in particular digits of the right hand can be best seen in Fig. 2 for males and Fig. 3 for females. However, the frequency distribution of flexion creases for the left hand was depicted in Fig. 4 for males and Fig. 5 for females. The incidence of extra + creases are extremely rare and generally restricted to the mid-phalangeal area of the digits. It has
On examination, the wrist shows radiocarpal joint swelling, with decreased range of motion. Flexion-extension is restricted, but pronosupination is full. Digital motion is without significant restriction.
His right-hand rests next to his hip in a fist form. With it rotated upward, having the back side of the fist pointing downward. The left hand has his finger pointed upward. His index to pinky finger extended fully (not hyperextended). The thumb is bent at the interphalangeal joint inward so it rests on/below the flange.
French physician, Antonie Marfan, discovered this disease in a 5 year old girl. The child had extremely long/thin fingers and arms that resembled a spider. Marfan’s patients shared some things in common. They all had long/thin arms and fingers, they were all thin, and they all had a tall height.
When he was young, Dylan Rosnick just wanted to play baseball, a simple enough request for a child growing up in the Loudoun County exurbs. He wanted to tie his shoes, too, and hold a pencil the right way, and button his shirt, and brush his teeth. There 's not a lot of guidance, though, for a child with Proteus syndrome, a genetic disorder that affects fewer than one in 1 million births worldwide, according to the National Institutes of Health. It causes overgrowth in bones, skin and other tissues.
The chin or mandibular symphysis can be analyzed by looking at the profile of the mandibule. The shape of dental arcade can be determined by looking at the skull from a ventral view and analyzing the shape that the upper teeth generate. The dentition can be determined by analyzing the size of the overall teeth with the size of the overall facial size. Finally, the retromolar space can be identified by evaluating the space between the last molar and the rest of the
Q. how is this disorder diagnosed & tested? A. its really self-diagnosed you don’t need to be a dr. to know if you have the disorder all you got to do is Look down at your hands a feet and check if you have an extra thumb, pinky or big/small toe. As for how its tests there are x-rays, enzyme tests, chromosome studies, and metabolic
Rett’s Syndrome Kelsey Leroux Child Development CYC 101 Lenore Simbulan October 14, 2016 Rett’s Syndrome Rett’s syndrome is defined by the Ontario Rett’s Syndrome Association (ORSA) as “a neurodevelopmental condition characterized by the loss of the spoken language and hand use, coupled with the development of distinctive hand stereotypies… It is usually caused by a mutation of the MECP2 gene on the x chromosome” (2016). Rett’s syndrome is considered by medical experts as a rare genetic neurological and developmental disorder that affects the way the brain develops causing a progressive inability to use muscles for speech, and eye and body movements. Most babies seem to develop normally until about six to eighteen months old. Rett’s
This is called the Nikolsky sign. (Porth’s, 2014). 7. Treatment Treatment of both TENS and SJS can involve surgical management and medical treatment. In both cases, the necrotic dermis is removed and a temporary skin covering is put into place to cover the open areas.
Because the lion was traditionally considered the king of the beasts and the eagle the king of birds, the gryphon was thought to be the king of all creatures. They are known for guarding treasure and were associated with gold; they were said to lay eggs in burrows on the grounds and these nests contained gold nuggets (Figure 12). Arthrogryposis is a congenital disorder characterized by joint contracture in two or more areas of the body. It derives its name from the Greek arthron (“joint”) and gryposis (“curved” or “hooked”) resembling to the hooked nails of gryphons. Children born with one or more joint contractures have abnormal fibrosis of the muscle tissue causing muscle shortening and joint ankyloses
The carpal tunnel functions as an entrance to the palm for many tendons and median nerve (1). The median nerve is one of the five branches that make up the brachial plexus. This nerve provides motor innervation to the flexor muscles of the forearm and hand as well as muscles responsible for movements of the thumb. It also provides sensory innervation to the volar aspects of the first three digits and half of the fourth digit, the palm, and the medial region of the forearm (2). Carpal tunnel syndrome (CTS) is a common painful disorder of the wrist and hand.
1. INTRODUCTION: The malady trigger finger earns its name from the painful popping or clicking sound elicited by flexion and extension of the involved digit. First described by Notta in 1850 [1], it is caused by a difference in diameters of a flexor tendon and its retinacular sheath due to thickening and narrowing of the sheath.
One such study by Faden et al, allowed the researchers to classify Erlenmeyer’s flask deformity into three groups. The first category describes the typical, common shape of the bone associated with EFD where a normal di-metaphyseal is not present. It is named EFD-T and it appears as a relatively normal trabecular (spongy) bone. This type of EFD is seen in frontometaphyseal dysplasia, craniometaphyseal dysplasia, craniodiaphyseal dysplasia, diaphyseal dysplasia-Engelmann type, metaphyseal dysplasia-Pyle type, Melnick–Needles osteodysplasty, and otopalatodigital syndrome type I. The second category describes the atypical type of EFD (EFD-A) as the bone lacks the normal modeling of the di-metaphyseal and an unusual appearance of the trabecular bone.
In this disorder, night time symptoms are common because a lot of people sleep with their wrists bent which makes the symptoms worse and causes severe pain. Also, when he plays the piano for an extended period of times, he is constantly flexing and extending his
The muscles of the affected limbs may be underdeveloped (hypoplastic), resulting in a tube-shaped limb with a soft, doughy
Much of this research has focused on the pattern formation that occurs in the limb along the antero-posterior axis in particular. In 1968, John Saunders and Mary Gasseling discovered a region of mesoderm on the posterior side of the limb bud known as the zone of polarizing activity (ZPA) (Saunders and Gasseling, 1968). When the posterior region of mesodermal cells was grafted to the anterior side of another chick limb bud, an amazing thing happened - a wing with a mirror-image pattern developed (Figure 3). The normal pattern of digits for a chick wing is 2-3-4 going anteriorly to posteriorly; the pattern of digits that developed in Saunders’ research was 2-3-4-4-3-2.