Shock
Definition
Shock is a medical emergency in which the organs and tissues of the body are not receiving an adequate flow of blood. This deprives the organs and tissues of oxygen (carried in the blood) and allows the buildup of waste products. Shock can result in serious damage or even death.
Description
There are three stages of shock: Stage I (also called compensated, or nonprogressive), Stage II (also called decompensated or progressive), and Stage III (also called irreversible).
In Stage I of shock, when low blood flow (perfusion) is first detected, a number of systems are activated in order to maintain/restore perfusion. The result is that the heart beats faster, the blood vessels throughout the body become slightly smaller in diameter, and the kidney works
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Causes and symptoms
Shock is caused by three major categories of problems: cardiogenic (meaning problems associated with the heart 's functioning); hypovolemic (meaning that the total volume of blood available to circulate is low); and septic shock (caused by overwhelming infection, usually by bacteria).
Cardiogenic shock can be caused by any disease, or event, which prevents the heart muscle from pumping strongly and consistently enough to circulate the blood normally. Heart attack, conditions which cause inflammation of the heart muscle (myocarditis), disturbances of the electrical rhythm of the heart, any kind of mass or fluid accumulation and/or blood clot which interferes with flow out of the heart can all significantly affect the heart 's ability to adequately pump a normal quantity of blood.
Hypovolemic shock occurs when the total volume of blood in the body falls well below normal. This can occur when there is excess fluid loss, as in dehydration due to severe vomiting or diarrhea, diseases which cause excess urination (diabetes insipidus, diabetes mellitus, and kidney failure), extensive burns, blockage in the intestine, inflammation of the pancreas (pancreatitis), or severe bleeding of
Title Day 1: I open my eyes, sitting up straight, only to realize that everything is turned 90 degree to the right… Next to us, a yellow school bus lies on its side. Pieces of glass scatters everywhere. Black smokes exit the cranky engine, traverse up to the height of an oak tree, then dissipate into the blue sky.
Revolutionary Heart is about a passionate advocate of the early women’s civil right movement, the temperance movement, the plight of the unprotected females and children, and the abolishment of slavery. Clarina H. Nichols was an inspiring, strong, maternal woman who crisscrossed the United States pushing for various reforms in the new frontier that impacted the lives of both men and woman who were unaware of the benefits of women’s citizenship and the responsibilities. She lived during the antebellum period and fought for her gender for decades until her death. She was an accomplished writer and delivered one hundred speeches over two decades defending her causes.
The Battlefield Heart and Open Wounds The three soldiers that were wounded during the Battle of Gettysburg were Robert E. Lee, Joshua L. Chamberlain, and John Buford. The wounds of each of these soldiers were Robert E. Lee had a heart disease, Joshua L. Chamberlain had a gunshot wound that went through both hips, and John Buford had a gunshot wound in his left arm. The main reason for the occurrence of the American Civil War was the controversy over slavery, this caused the division of the U.S. and formed the North and South. The South allowed slavery while the North didn’t.
Sepsis is the leading cause of shock and circulatory collapse, which can ultimately lead to death. (Conrad, 2015) Symptoms for sepsis include a temperature, a rapid heartbeat, fast breathing, dizziness, confusion or disorientation, and a reduced urine flow. (Healthdirect.gov.au, 2015) The spread of the Staphylococcus aureus bacteria to the heart can lead to an infection of the heart valves, known as endocarditis, which results in heart failure.
While these efforts have substantially limited the amount of patients who receive the shock-treatment to less than a third, parents of these continue to defend the practice because they feel as if there are no alternative treatments for their
The learner, or victim, is actually an actor who receives no shock at all. The point of the experiment is to see how far a person will go in a situation where he is ordered to inflict increasing pain on a protesting victim. Milgram, during his study, noted when a conflict occurred, ”At 75 volts, he grunts; at 120 volts, he complains loudly; at 150, he demands to be released from the experiment. As the voltage increases, his protests become more vehement and emotional. At 285 volts, his response can be described only as an agonized scream.
The shock started from 15 volts and increased by 15 per wrong answer up a maximum shock of 450 volts (Milgram Experiment-Obedience to Authority, 2015). In Milgram’s first experiment, fifteen out of the forty participants refused to continue at some point in the experiment, while 25 participants continued all the way to 450 volts, “shocking” the learner three times before the acting scientist ended the experiment. The teachers, however, did not know that there were no shocks and the procedure was perfectly safe. To sell the fact that the learners were hurt, the scientist would bring out the acting learner whose face was “covered in tears and looked haggard” to meet one of the participants, Joseph Dimow. The actor “thanked” him for stopping the experiment and noted the anticipation was worse than the shock
The shock is intended to be non-lethal, but sufficient to temporarily stun an
This may occur during medical procedures, such as when blood is being drawn from a vein. • Common activities, such as coughing, swallowing, stretching, or going to the bathroom. • Emotional stress. • Being in a confined space.
The third thing i learned was the progression of shock. When checking for shock, shock itself can not be seen, but the signs and symptoms can be. Such as, blood loss, which is called compensated shock. Decompesated shock, is the last step, and that is when the blood pressure drops and after that, there 's not much you can do. Because after that shock is terminal which means there is no saving someone 's life.
His hoarse gasps for oxygen, distinct against the background of gunfire. Time seemed to slow down as he collapsed onto all fours, foaming green froth from the corners of his mouth as he was coughing and spitting a pool of blood, dirt and saliva. He lifted his head to make a final attempted to stand up and get to safety but his body could take no more and he slumped backward face up into the dirt. His eyes wide open, with the look of agony etched across his
A Transient Ischemic Attack, also known as a mini stroke, is an acute illness where blood flow to a certain part of the brain gets blocked which most of the time is caused by a blood clot (Webmd n.d.). After a couple minutes the clot dissolves, blood flow continues again and symptoms disappear. It is called a mini stroke because all the signs and symptoms are the same as a stroke, but the symptoms last a shorter period of time and there is no permanent damage done to the cells. According to the website mayoclinic.org, About 1 in 3 people who have a transient ischemic attack will eventually have a stroke, with half occurring within a year after the transient ischemic attack (Mayo Clinic Staff 2014). Overall, even though it is considered a mini stroke and the symptoms last shorter time, it is still a serious condition and need medical attention immediately.
Normal shock front The air passes through the tube slower than the speed the tube passes through the surrounding air. As a consequence, air
Bleeding from the nose and mouth 2. Shock due to excessive loss of blood Diagnosis Diagnosis is done with the help of observed symptoms.
Abstract Introduction Small heat shock proteins (sHSPs) are a family of molecular chaperones which play an important role in mitigating and preventing the aggregation of misfolded proteins under conditions of stress such as hypoxia or heat and thereby contribute to maintaining intracellular homeostasis. These properties are exploited in many organs, including the heart where they are thought to play a crucial role in the cardiac hypertrophy. Cardiac hypertrophy is a condition in which the heart compensates for a pressure overload caused by several cardiovascular diseases. However, cardiac hypertrophy itself poses a health risk as it increases the risk of severe cardiovascular events. Clarifying the role of sHSPs in normal as well as hypertrophic condition could be important in understanding the underlying mechanism of sHSPs in heart functioning.