Type 2 DM results from association between hereditary, natural and behavioural risk factor. Type 2 DM is due primarily to lifestyle factors and genetics. A number of lifestyle factors are known to be important to the development of type 2 DM. These are physical inactivity, sedentary lifestyle, cigarette smoking and generous consumption of alcohol (Abdulfatai B. Olokoba, 201).
Her BP and HR were elevated, and I did not have any PRN order for antihypertensive drug whatsoever. She was desaturating, thus I titrated the O2 from 2 L to 6 L, her O2 went back to baseline. However. My patient stated that her pain was increasing and asked me if she could have the same med that was given previously in the ED (sublingual nitro) for her back
Pharmacologic treatments for diastolic heart failure and systolic heart failure are similar in the fact they both should include an ACEI, an ARB, beta blocker, and a diuretic. The following dosages and instructions are available for both heart failures. For ACEI, the treatment should be initiated at low dosages and slowly titrated upward if the patient tolerates them. Captopril should be taken by mouth on an empty stomach (at least 1 hour before meals) usually two to three times a day starting at 6.25 mg initially then increasing to 50 mg three times a day. If a patient is unable to tolerate an ACEI, an ARB would be beneficial because they do not create the same reaction.
E.g. 99mTc-tetrofosmin (Myoview, GE healthcare), 99mTc-sestamibi (Cardiolite, Bristol-Myers Squibb now Lantheus Medical Imaging). Following this, the heart rate is raised to induce myocardial stress, either by exercise or pharmacologically with adenosine, dobutamine or dipyridamole (aminophylline can be used to reverse the effects of
While auscultating sounds of lung fields no wheezing was found, and VS were within normal range for patient as determined through comparison of chartings on 10/23/2015 thru the morning and lunch VS of 10/26/ 2015 before impaired gas exchange was detected. 10/26/2015 2. Administer O2 @ 2L N/C
2012). An anticholinergic nebulizer, ipratropium was given to mrs.Smith as per the order which helped to reduce dyspnea and cough slightly. Her Early warning score still remains 6. On detailed examination, Ed doctor suspected mrs.Smith may be having heart failure. ECG done on her which shows sinus tachycardia.
(NSW Health 2012:20). Glyceryl Trinitrate acts on vascular smooth muscles and dilate arteries and veins. Vasodilatation results in the reduction of venous return which also reduces the work load of the heart and oxygen demand. GTN sublingual tablet or spray can be repeated every 5 minute for maximum of 3 doses. Regular monitoring of vital signs is important because GTN can cause Hypotension.
Christina Markevich 10/29/2015 Congestive Heart Failure What is Congestive Heart Failure? Congestive Heart Failure is something that occurs when the heart is no longer able to pump enough blood to the rest of the body, or when is just is not able to pump blood as well as it should. Some people happen to have either of these problems, some people have both.
Patient’s primary care doctor stopped her oral medication due to nausea and vomiting; and her liver function tests were elevated. She does not know names, but the sleep aid and the oral medication for pain during the day were stopped. She tolerates the medications well. Patient shows no evidence of developing medication dependency. Patient does not feel the current medication she is taking adequately addressing her pain needs and would like to try a different medication.
Short acting anticholinergics are not considered relief medications because they normally take longer to work than our beta-agonist medications. Though the lists of short acting anticholinergics are short these medications are great for people with COPD. They have fewer side effects than beta-agonist drugs and they are good bronchodilators. The long acting anticholinergic drug takes about 20 minutes to work and can last up to 24 hours, although it is thought of as a “reliever” it’s long lasting effects would make it an excellent maintenance drug for patients with COPD
This compound has reached preclinical studies and has shown to reduce pulmonary capillary pressure, systemic and renal vascular resistance and increased cardiac output and is only active when the renin-angiotensin system is active (for example in acute heart failure). Scientists suggested that
Basic pharmacology of agent used in the treatment of asthma Pharmacodynamics about Salbutamol (INN) or albuterol (USAN), a moderately selective beta (2)-receptor agonist similar in structure to terbutaline, is widely used as a bronchodilator to manage asthma and other chronic obstructive airway diseases. The R-isomer, levalbuterol, is responsible for bronchodilation while the S-isomer increases bronchial reactivity. The R-enantiomer is sold in its pure form as Levalbuterol. The manufacturer of levalbuterol, Sepracor, has implied (although not directly claimed) that the presence of only the R-enantiomer produces fewer side-effects. Mechanism of action Salbutamol is a beta (2)-adrenergic agonist and thus it stimulates beta (2)-adrenergic receptors.